Downregulation of LINC00638 contributes to the pathogenesis of rheumatoid arthritis-associated interstitial lung disease via inhibiting the Nrf2/ARE signaling pathway

LIAO Zhuojun; TANG Naiwang; CHEN Jiahui; SUN Xueying; LU Jiamin; WU Qin; YU Ronghuan; ZHOU Ying

doi:10.12025/j.issn.1008-6358.2025.20241417

Chinese Journal Of Clinical Medicine, Volume. 32, Issue 3, 421(2025)

Downregulation of LINC00638 contributes to the pathogenesis of rheumatoid arthritis-associated interstitial lung disease via inhibiting the Nrf2/ARE signaling pathway

LIAO Zhuojun^1,2, TANG Naiwang^3、*, CHEN Jiahui², SUN Xueying², LU Jiamin², WU Qin³, YU Ronghuan³, and ZHOU Ying²

Author Affiliations

¹Department of Rheumatology and Immunology, Shanghai Xuhui Central Hospital, Shanghai 200031, China

²Department of General Practice, Shanghai Xuhui Central Hospital, Shanghai 200031, China

³Department of Respiratory and Critical Care Medicine, Shanghai Xuhui Central Hospital, Shanghai 200031, China

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LIAO Zhuojun, TANG Naiwang, CHEN Jiahui, SUN Xueying, LU Jiamin, WU Qin, YU Ronghuan, ZHOU Ying. Downregulation of LINC00638 contributes to the pathogenesis of rheumatoid arthritis-associated interstitial lung disease via inhibiting the Nrf2/ARE signaling pathway[J]. Chinese Journal Of Clinical Medicine, 2025, 32(3): 421

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Paper Information

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Received: Dec. 9, 2024

Accepted: Aug. 22, 2025

Published Online: Aug. 22, 2025

The Author Email: TANG Naiwang (tangnw88@hotmail.com)

DOI:10.12025/j.issn.1008-6358.2025.20241417

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