Flavour Fragrance Cosmetics, Volume. , Issue 3, 43(2025)
Mechanism of Formation and Skin Care Solutions for Oily and Sensitive Skin
[1] [1] BAUMANN L. Understanding and treating various skin types: the baumann skin type indicator [J]. Dermatologic Clinics, 2008, 26(3): 359-373.
[2] [2] MISERY L, STNDER S, SZEPIETOWSKI J C,et al. Definition of sensitive skin: an expert position paper from the special interest group on sensitive skin of the international forum for the study of itch [J]. Acta Dermato-Venereologica, 2017, 97(1): 4-6.
[4] [4] SAKUMA T H, MAIBACH H I. Oily skin: an overview [J]. Skin Pharmacology and Physiology, 2012, 25(5): 227-235.
[5] [5] ENDLY D C, MILLER R A. Oily skin: a review of treatment options [J]. The Journal of Clinical and Aesthetic Dermatology, 2017, 10(8): 49-55.
[6] [6] HONG J Y, PARK S J, SEOSEO S J,et al. Oily sensitive skin: a review of management options [J]. Journal of Cosmetic Dermatology, 2020, 19(5): 1016-1020.
[8] [8] THIBOUTOT D, BAYNE E, THORNE J,et al. Immunolocalization of 5-reductase isozymes in acne lesions and normal skin [J]. Archives of Dermatology, 2000, 136(9): 1125-1129.
[9] [9] HUNKELER M, HAGMANN A, STUTTFELD E,et al. Structural basis for regulation of human acetyl-CoA carboxylase [J]. Nature, 2018, 558(7710): 470-474.
[10] [10] ABU-ELHEIGA L, MATZUK M M, KORDARI P,et al. Mutant mice lacking acetyl-CoA carboxylase 1 are embryonically lethal [J]. Proceedings of the National Academy of Sciences of the United States of America, 2005, 102(34): 12011-12016.
[12] [12] ESLER W P, TESZ G J, HELLERSTEIN M K,et al. Human sebum requiresde novolipogenesis, which is increased in acne vulgaris and suppressed by acetyl-CoA carboxylase inhibition[J]. Science Translational Medicine, 2019, 11(492): eaau8465.
[15] [15] BOLLAG W B, AITKENS L, WHITE J,et al. Aquaporin-3 in the epidermis: more than skin deep [J]. American Journal of Physiology. Cell Physiology, 2020, 318(6): C1144-C1153.
[17] [17] CHIEOSILAPATHAM P, KIATSURAYANON C, UMEHARA Y,et al. Keratinocytes: innate immune cells in atopic dermatitis[J]. Clinical & Experimental Immunology, 2021, 204(3): 296-309.
[18] [18] DE BENEDETTO A, RAFAELS N M, MCGIRT L Y,et al. Tight junction defects in patients with atopic dermatitis [J]. Journal of Allergy and Clinical Immunology, 2011, 127(3): 773-786.e7.
[23] [23] WOHLRAB J, GEBERT A, NEUBERT R H H. Lipids in the skin and pH [M]// pH of the skin: issues and challenges. Basel, Switzerland: Karger AG, 2018: 64-70.
[24] [24] GRELLNER W. Time-dependent immunohistochemical detection of proinflammatory cytokines (IL-1, IL-6, TNF-) in human skin wounds [J]. Forensic Science International, 2002, 130(2/3): 90-96.
[25] [25] KEE J Y, JEON Y D, KIM D S,et al. Korean Red Ginseng improves atopic dermatitis-like skin lesions by suppressing expression of proinflammatory cytokines and chemokinesin vivoandin vitro[J]. Journal of Ginseng Research, 2017, 41(2): 134-143.
[26] [26] DUBIN C, DEL DUCA E, GUTTMAN-YASSKY E. The IL-4, IL-13 and IL-31 pathways in atopic dermatitis [J]. Expert Review of Clinical Immunology, 2021, 17(8): 835-852.
[27] [27] BORDA L J, PERPER M, KERI J E. Treatment of seborrheic dermatitis: a comprehensive review [J]. Journal of Dermatological Treatment, 2019, 30(2): 158-169.
[28] [28] PIQUERO-CASALS J, HEXSEL D, MIR-BONAF J F,et al. Topical non-pharmacological treatment for facial seborrheic dermatitis [J]. Dermatology and Therapy, 2019, 9(3): 469-477.
[29] [29] TRZNADEL-GRODZKA E, BASZKOWSKI M, ROTSZTEJN H. Investigations of seborrheic dermatitis. Part I. The role of selected cytokines in the pathogenesis of seborrheic dermatitis[J]. Postepy Higieny i Medycyny Doswiadczalnej, 2012, 66: 843-847.
[30] [30] JAPPE U, INGHAM E, HENWOOD J,et al.Propionibacterium acnesand inflammation in acne;P. acneshas T-cell mitogenic activity [J]. British Journal of Dermatology, 2002, 146(2): 202-209.
[31] [31] KIM J, OCHOA M T, KRUTZIK S R,et al. Activation of toll-like receptor 2 in acne triggers inflammatory cytokine responses [J]. Journal of Immunology, 2002, 169(3): 1535-1541.
[34] [34] STEINHOFF M, STNDER S, SEELIGER S,et al. Modern aspects of cutaneous neurogenic inflammation [J]. Archives of Dermatology, 2003, 139(11): 1479-1488.
[35] [35] MACK K, FISCHER M J M. Disrupting sensitization of TRPV4 [J]. Neuroscience, 2017, 352: 1-8.
[37] [37] SHIMOJO N, JESMIN S, ZAEDI S,et al. Contributory role of VEGF overexpression in endothelin-1-induced cardiomyocyte hypertrophy [J]. American Journal of Physiology. Heart and Circulatory Physiology, 2007, 293(1): H474-H481.
[39] [39] VERHOEVEN E W M, DE KLERK S, KRAAIMAAT F W,et al. Biopsychosocial mechanisms of chronic itch in patients with skin diseases: a review [J]. Acta Dermato-Venereologica, 2008, 88(3): 211-218.
[41] [41] GRIGORE O, MIHAILESCU A I, SOLOMON I,et al. Role of stress in modulation of skin neurogenic inflammation [J]. Experimental and Therapeutic Medicine, 2019, 17(2): 997-1003.
[42] [42] PAUS R, THEOHARIDES T C, ARCK P C. Neuroimmunoendocrine circuitry of the “brain-skin connection” [J]. Trends in Immunology, 2006, 27(1): 32-39.
[43] [43] LYU F, WU T, BIAN Y Z,et al. Stress and its impairment of skin barrier function [J]. International Journal of Dermatology, 2023, 62(5): 621-630.
[45] [45] SCHMID-WENDTNER M H, KORTING H C. The pH of the skin surface and its impact on the barrier function [J]. Skin Pharmacology and Physiology, 2006, 19(6): 296-302.
[46] [46] TANAKA A, MATSUDA A, JUNG K,et al. Ultra-pure soft water ameliorates atopic skin disease by preventing metallic soap deposition in NC/Tnd mice and reduces skin dryness in humans [J]. Acta Dermato-Venereologica, 2015, 95(7): 787-791.
[51] [51] ABDEL-MOTALEB A A, ABV-DTEF E E, HVSSEIN M R. Dermal morphological changes following salicylic acid peeling and microdermabrasion [J]. Journal of Cosmetic Dermatology, 2017, 16(4): e9-e14.
[58] [58] SALMINEN S, COLLADO M C, ENDO A,et al. Reply to: postbiotics: when simplification fails to clarify [J]. Nature Reviews Gastroenterology & Hepatology, 2021, 18: 827-828.
[61] [61] RAMAZANI E, AKABERI M, AHMAD EMAMI S,et al. Pharmacological and biological effects of alpha-bisabolol: an updated review of the molecular mechanisms [J]. Life Sciences, 2022, 304: 120728.
[62] [62] BRAGA P C, DAL SASSO M, FONTI E,et al. Antioxidant activity of bisabolol: inhibitory effects on chemiluminescence of human neutrophil bursts and cell-free systems [J]. Pharmacology, 2009, 83(2): 110-115.
[63] [63] LI G X, WU H Y, SUN L Q,et al. (-)--bisabolol alleviates atopic dermatitis by inhibiting MAPK and NF-B signaling in mast cell [J]. Molecules, 2022, 27(13): 3985.
[64] [64] LEITE C D S, BONAF G A, PIRES O C,et al. Dipotassium glycyrrhizininate improves skin wound healing by modulating inflammatory process [J]. International Journal of Molecular Sciences, 2023, 24(4): 3839.
Get Citation
Copy Citation Text
SUN Ruirui, ZHANG Mingjie, ZHANG Yexiang, QI Fengwei. Mechanism of Formation and Skin Care Solutions for Oily and Sensitive Skin[J]. Flavour Fragrance Cosmetics, 2025, (3): 43
Category:
Received: May. 29, 2024
Accepted: Aug. 26, 2025
Published Online: Aug. 26, 2025
The Author Email: